Recent Publications from Members of the BIIC

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The list of publications was last updated August 26th, 2009. If you would like to add another page, email Joshua Gray with an Endnote-formatted file containing the references you would like to add. Alternatively, you may request permission to add references and, once verified as a BIIC member, you will be able to submit them yourself.

You are hereRecent Publications of Members of the Boston Ithaca Islet Club

Recent Publications of Members of the Boston Ithaca Islet Club

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Filters: Author is Poitout, V.  [Clear All Filters]
2009
Peyot ML, Guay C, Latour MG, Lamontagne J, Lussier R, Pineda M, Ruderman NB, Haemmerle G, Zechner R, Joly E, Madiraju SR, Poitout V, Prentki M.  2009.  Adipose triglyceride lipase is implicated in fuel- and non-fuel-stimulated insulin secretion. J Biol Chem. 284(25):16848-59. Abstract
Guo S, Burnette R, Zhao L, Vanderford NL, Poitout V, Hagman DK, Henderson E, Ozcan S, Wadzinski BE, Stein R.  2009.  The stability and transactivation potential of the mammalian MafA transcription factor are regulated by serine 65 phosphorylation. J Biol Chem. 284(2):759-65. Abstract
Lamontagne J, Pepin E, Peyot ML, Joly E, Ruderman NB, Poitout V, Madiraju SR, Nolan CJ, Prentki M.  2009.  Pioglitazone acutely reduces insulin secretion and causes metabolic deceleration of the pancreatic beta-cell at submaximal glucose concentrations. Endocrinology. 150(8):3465-74. Abstract
2008
Kebede M, Alquier T, Latour MG, Semache M, Tremblay C, Poitout V.  2008.  The fatty acid receptor GPR40 plays a role in insulin secretion in vivo after high-fat feeding. Diabetes. 57(9):2432-7. Abstract
Poitout V.  2008.  Glucolipotoxicity of the pancreatic beta-cell: myth or reality? Biochem Soc Trans. 36(Pt 5):901-4. Abstract
Poitout V, Robertson RP.  2008.  Glucolipotoxicity: fuel excess and beta-cell dysfunction. Endocr Rev. 29(3):351-66. Abstract
Hagman DK, Latour MG, Chakrabarti SK, Fontes G, Amyot J, Tremblay C, Semache M, Lausier JA, Roskens V, Mirmira RG, Jetton TL, Poitout V.  2008.  Cyclical and alternating infusions of glucose and intralipid in rats inhibit insulin gene expression and Pdx-1 binding in islets. Diabetes. 57(2):424-31. Abstract
Poitout V.  2008.  Phospholipid hydrolysis and insulin secretion: a step toward solving the Rubik's cube. Am J Physiol Endocrinol Metab. 294(2):E214-6.
2007
Madiraju SR, Poitout V.  2007.  G protein-coupled receptors and insulin secretion: 119 and counting. Endocrinology. 148(6):2598-600.
Latour MG, Alquier T, Oseid E, Tremblay C, Jetton TL, Luo J, Lin DC, Poitout V.  2007.  GPR40 is necessary but not sufficient for fatty acid stimulation of insulin secretion in vivo. Diabetes. 56(4):1087-94. Abstract
2006
Metz TO, Jacobs JM, Gritsenko MA, Fontes G, Qian WJ, Camp D. G. 2, Poitout V, Smith RD.  2006.  Characterization of the human pancreatic islet proteome by two-dimensional LC/MS/MS. J Proteome Res. 5(12):3345-54. Abstract
Poitout V, Hagman D, Stein R, Artner I, Robertson RP, Harmon JS.  2006.  Regulation of the insulin gene by glucose and fatty acids. J Nutr. 136(4):873-6. Abstract
Poitout V.  2006.  [2006 Apollinaire Bouchardat Award. Fatty acids and pancreatic beta cell function]. Journ Annu Diabetol Hotel Dieu. :37-46.
2005
Hagman DK, Hays LB, Parazzoli SD, Poitout V.  2005.  Palmitate inhibits insulin gene expression by altering PDX-1 nuclear localization and reducing MafA expression in isolated rat islets of Langerhans. J Biol Chem. 280(37):32413-8. Abstract
Pino MF, Ye DZ, Linning KD, Green CD, Wicksteed B, Poitout V, Olson LK.  2005.  Elevated glucose attenuates human insulin gene promoter activity in INS-1 pancreatic beta-cells via reduced nuclear factor binding to the A5/core and Z element. Mol Endocrinol. 19(5):1343-60. Abstract
Zhao L, Guo M, Matsuoka TA, Hagman DK, Parazzoli SD, Poitout V, Stein R.  2005.  The islet beta cell-enriched MafA activator is a key regulator of insulin gene transcription. J Biol Chem. 280(12):11887-94. Abstract
Bihan H, Rouault C, Reach G, Poitout V, Staels B, Guerre-Millo M.  2005.  Pancreatic islet response to hyperglycemia is dependent on peroxisome proliferator-activated receptor alpha (PPARalpha). FEBS Lett. 579(11):2284-8. Abstract